“As a young girl, living in rural Manitoba, Canada, my brother would harvest wild rabbits for our winter protein. When my brother was skinning the rabbits, he was extremely careful to check for bubbles under the skin and in the meat. If he found bubbles, he knew that the rabbits had a virus, due to over population. We were not able to eat those rabbits that year. . . and would have to wait until nature had taken its course. My point being, there is no 5G or anything that would support your theory.” The writer also describes TB among the northern Inuit, “which had taken hold, due to living in crowded conditions and of course eating the foods that the Europeans had introduced to them. Killing people off with fast foods and the likes has brought us to where we are. This Covid virus is just the beginning of the major viruses that are just around the corner.” She also referred to very crowded and filthy living conditions in China, presumably in connection with diseases like cholera.
The rabbit disease to which she refers is called myxomatosis and since I am an English major, I know about this disease from a famous poem by the British poet Philip Larkin. He wrote Myxomatosis in response to the introduction of the myxomatosis disease to the wild rabbit population of Britain during the 1950s. The poem describes what happens when Larkin encounters a sick rabbit and kills it with a stick. He puts the animal out of its misery instead of letting it slowly die of the disease.
Caught in the center of a soundless field
While hot inexplicable hours go by
What trap is this? Where were its teeth concealed?
You seem to ask.
I make a sharp reply,
Then clean my stick. I’m glad I can’t explain
Just in what jaws you were to suppurate:
You may have thought things would come right again
If you could only keep quite still and wait.
But I digress. . .
The official view is that a virus carried in the saliva of fleas, mosquitoes, and other insects causes myxomatosis, with overcrowded populations the most vulnerable to illness. Symptoms include swelling at the site of the “infection”—the insect bite—followed by fever, swelling in other areas (eyelids, face, base of ears, anogenital area), skin lesions, ocular and nasal discharge, respiratory distress, hypothermia, closure of the eyelids due to swelling, and death. These are some very miserable rabbits!
A typical study cited for a viral cause of myxomatosis is “The pathogenesis of infectious myxomatosis; the mechanism of infection and the immunological response in the European rabbit (Oryctolagus cuniculus),” published in the British Journal of Experimental Pathology, 1953. The researchers were able to make rabbits display the symptoms of myxomatosis including death by injecting them with “virulent myxomatosis.” The recipe for this witches’ brew includes ground up organs and heated blood of sick flea-bitten rabbits, “passed through” other rabbits, which are then bled to death to obtain the serum, then grown on chicken embryos. Healthy rabbits injected—often several times—with this “virulent myxomatosis” usually do sicken and die.
The Wikipedia entry for “myxomatosis” includes a discussion of how to diagnose this so-called viral disease, hinting at the problems involved. According to the article, usually the basis of a myxomatosis diagnosis follows a description of the characteristic clinical appearance—in other words, you can tell by the symptoms. For further confirmation, “researchers have turned to histopathology, electron microscopy, and virus isolation. . . Histopathologic examination of affected skin typically shows undifferentiated mesenchymal cells within a matrix of mucin, inflammatory cells, and edema. Intracytoplasmic inclusions may be seen in the epidermis and in conjunctival epithelium.” In other words, they don’t see isolated virus, just messed up cells.
Negative-stain electron microscopic examination allows “rapid visualization of poxviruses, but does not allow specific verification of virus species or variants.”
“Virus isolation remains the ‘gold standard’ against which other methods of virus detection are compared.” This is true, virus isolation is the gold standard. “Theoretically at least, a single viable virus present in a specimen can be grown in cultured cells, thus expanding it to produce enough material to permit further detailed characterization.” Theoretically, yes, but in practice, pure virus introduced into animals or animal cells has little effect. Only “virulent virus” will appear to multiply and cause disease.
That’s why scientists use “The more recent development of molecular methods such as polymerase chain reaction (PCR) and real-time polymerase chain reaction assays [for] faster and more accurate methods of myxoma virus identification. Real time PCR simplifies the diagnosis of myxomatosis by allowing nasal, ocular, or genital swabs to be quickly tested.” The problem is that PCR does not identify specific viruses, only snippets of genetic material. So while this method may be fast, it is certainly not “more accurate.”
Even if scientists do isolate the pure virus, they still need to show that this pure virus can make healthy rabbits sick. During the 1950s, myxomatosis was intentionally introduced in Australia, France and Chile to control wild European rabbit populations. Brought to these countries to serve as a food source in the eighteenth and nineteenth centuries, and having few enemies, these rabbits bred like. . . like rabbits. . . and soon overwhelmed the countryside, eating every green thing in sight.
Did scientists kill them off by introducing pure isolated virus or even “virulent” virus into the rabbits? No, they introduced fleas. The fleas dutifully bit the rabbits and myxomatosis followed, killing off huge numbers.
Blood-sucking insects like fleas, mosquitoes and ticks contain an enzyme called apyrase in their saliva, which prevents platelet aggregation (clotting) at the site of the bite. Apyrase keeps the blood liquid until the insect has had its fill. The enzyme can overwhelm blood-clotting capabilities and act as a poison in an animal that is breathing bad air in overcrowded warrens, undernourished due to scarce food (including clot-promoting vitamin K in green fodder) and then bitten many times. The point is that you don’t need an “infectious virus” to explain myxomatosis. Fleas and mosquitoes are one of nature’s ways of controlling overpopulation in various species of animals, and they do it by poisoning them.
Are researchers seeing “viruses” in their swabs and isolates, or helpful exosomes which multiply in situations of stress and disease?
Likewise, you don’t need to call on “infectious viruses” to explain human diseases like TB in the Inuit or cholera among the Chinese. Nutrient deficiencies, crowding and filth are perfectly capable of causing suffering and death without the help of “viruses.” Blaming a virus just encourages scientists and public health officials to focus on drugs and vaccines as a solution, instead of the tedious and unglamorous work of providing better food, clean water and a sanitary environment.